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- Academic Journals (23)
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- Search Terms:ISSN: 18661947AndISSN: 18661955AndVolume Number: 4AndIssue Number: 1AndStart Page: 25AndDate: 2012 Revise Search
- 1From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Published structural neuroimaging studies of prenatal cocaine exposure (PCE) in humans have yielded somewhat inconsistent results, with several studies reporting no significant differences in brain...
- 2From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedInfant sibling studies have been at the vanguard of autism spectrum disorders (ASD) research over the past decade, providing important new knowledge about the earliest emerging signs of ASD and expanding our...
- 3From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Autism spectrum disorders and schizophrenia have been associated with an overlapping set of copy number variant loci, but the nature and degree of overlap in copy number variants (deletions compared to...
- 4From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedThis thematic series presents theoretical and empirical papers focused on understanding autism from the perspective of reward processing deficits. Although the core symptoms of autism have not traditionally been...
- 5From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Exposure to alcohol in utero is a known cause of mental retardation. Although a certain degree of motor impairment is always associated with fetal alcohol spectrum disorder, little is known about the...
- 6From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground One hypothesis for the social deficits that characterize autism spectrum disorders (ASD) is diminished neural reward response to social interaction and attachment. Prior research using established monetary...
- 7From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Alcohol related neurodevelopmental disorder (ARND) falls under the umbrella of fetal alcohol spectrum disorder (FASD), but individuals do not demonstrate the facial characteristics associated with fetal...
- 8From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedThis review summarizes evidence of dysregulated reward circuitry function in a range of neurodevelopmental and psychiatric disorders and genetic syndromes. First, the contribution of identifying a core mechanistic...
- 9From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Increased prenatal testosterone exposure has been hypothesized as a mechanism underlying autism spectrum disorders (ASD). However, no studies have prospectively measured prenatal testosterone exposure and...
- 10From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Chromosome 22q11.2 Deletion Syndrome (22q11.2DS) occurs in approximately 1:4,000 live births with a complex and variable presentation that includes medical, socioemotional and psychological symptoms with...
- 11From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedPurpose There is increasing evidence that neurodevelopmental differences in people with Fragile X syndrome (FraX) may be explained by differences in glutamatergic metabolism. Premutation carriers of FraX were...
- 12From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedMost behavioral training regimens in autism spectrum disorders (ASD) rely on reward-based reinforcement strategies. Although proven to significantly increase both cognitive and social outcomes and successfully reduce...
- 13From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Many children who are late talkers go on to develop normal language, but others go on to have longer-term language difficulties. In this study, we considered which factors were predictive of persistent...
- 14From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Individuals with Autism Spectrum Disorders (ASD) typically show impaired eye contact during social interactions. From a young age, they look less at faces than typically developing (TD) children and tend...
- 15From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedThe combination of economic games and human neuroimaging presents the possibility of using economic probes to identify biomarkers for quantitative features of healthy and diseased cognition. These probes span a range of...
- 16From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedThe autism spectrum disorders (ASDs) arise from a diverse array of genetic and environmental origins that disrupt the typical developmental trajectory of neural connectivity and synaptogenesis. ASDs are marked by...
- 17From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground Williams syndrome (WS) is a rare genetic disorder caused by the deletion of approximately 25 genes at 7q11.23 that involves mild to moderate intellectual disability (ID). When using functional magnetic...
- 18From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground A previous study reported enhanced psychomotor speed, and subtle but significant cognitive impairments, modulated by age and by mutations in the fragile X mental retardation 1 (FMR1) gene in adult female...
- 19From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedBackground People with autism have abnormal preferences, ranging from an apparent lack of preference for social stimuli to unusually strong preferences for restricted sets of highly idiosyncratic stimuli. Yet the...
- 20From: Journal of Neurodevelopmental Disorders. (Vol. 4, Issue 1) Peer-ReviewedFor decades, researchers have sought to clarify the nature of the social communication impairments in autism, highlighting impaired or atypical 'social attention' as a key measurable construct that helps to define the...