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- 1From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedWe conducted a prospective study in pediatric patients presenting to an emergency center (EC) to (1) test the ability of urinary acute kidney injury (AKI) biomarkers to predict AKI presence and severity and (2)...
- 2From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedSteroid-resistant nephrotic syndrome (NS) remains one of the most intractable causes of end-stage renal disease in the first two decades of life. Several genes have been involved including NPHS1, NPHS2, WT1, PLCE1, and...
- 3From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedArterial stiffness is an increasingly recognized independent predictor of cardiovascular morbidity. Vessel volume and wall texture are the main determinants of pulse wave velocity (PWV), the most commonly used indicator...
- 4From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-Reviewed
Behavioral disorders and low quality of life in children and adolescents with chronic kidney disease
Recent years has seen an increasing interest in the quality of life (QOL) of children with chronic kidney disease (CKD). The objective of this cross-sectional study was to investigate the prevalence of behavioral... - 5From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedMigration implies differences in lifestyle, dietary and health behavior practice, and adherence, all of which are relevant factors in terms of disease outcome. However, renal transplantation in immigrant groups has been...
- 6From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedCystinosis is the major cause of inherited Fanconi syndrome, and should be suspected in young children with failure to thrive and signs of renal proximal tubular damage. The diagnosis can be missed in infants, because...
- 7From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedResponse Diverse factors may lead to early graft dysfunction in pediatric renal transplantation, including acute tubular necrosis, antibody-mediated rejection, severe accelerated cellular rejection, nephrotoxicity...
- 8From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedSirs, Persistent secondary hyperoxaluria has been reported in few patients affected by distal renal tubular acidosis (dRTA) [1]. Hyperoxaluria is a genetic or acquired metabolic disorder. The patients affected by...
- 9From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedThe production of free radicals can cause renal injury and play an important role in the pathogenesis of idiopathic nephrotic syndrome. Markers of reactive oxygen species (ROS) were evaluated in 48 patients with active...
- 10From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedAcute idiopathic tubulointerstitial nephritis (TIN) is considered a condition with a good long-term prognosis. However, there is evidence that some patients develop permanent renal impairment. The aim of this study was...
- 11From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedNephronophthisis (NPHP) is an autosomal recessive cystic kidney disease and the most frequent genetic cause of end-stage renal disease up to the third decade of life. It is caused by mutations in 11 different genes,...
- 12From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedOur recent in vitro study demonstrated peroxisome proliferator-activated receptor-[gamma] (PPAR-[gamma]) agonist potentiated the anti-inflammatory effect of angiotensin receptor blocker (ARB) in tubular epithelial cell...
- 13From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedAcute poststreptococcal glomerulonephritis (APSGN) is the most common form of postinfectious nephritis worldwide. The relationship between inflammation and arterial stiffness has been described elsewhere, but there have...
- 14From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedThe international and particularly the German community of pediatricians and pediatric nephrologists are mourning, because it has lost an eminent colleague, scientist, and clinician, Lothar Bernd Zimmerhackl, who died...
- 15From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedRenal Dysfunction affects 5-18% of patients with sickle cell disease (SCD). To date, no studies have described urinary levels of transforming growth factor [beta]-1 (TGF-[beta]1), a marker of fibrosis, and neutrophil...
- 16From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedThe aim of this study was to assess the relationship between cystatin C (CysC), cardiometabolic risk factors (CMRFs), and body composition in severely obese children. We evaluated 117 children aged 7-14 years old....
- 17From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedGrowth failure is common in children with chronic kidney disease, and successful treatment is a major challenge in the management of these children. The aetiology is multi-factorial with "chronic kidney...
- 18From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedDenys-Drash syndrome (DDS) consists of the triad of nephropathy, male pseudohermaphroditism, and Wilms tumor caused by mutations within exons 8 or 9 of the Wilms tumor suppressor gene 1. Early onset nephrotic syndrome...
- 19From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedCase summary In 2008, an 11-year-old girl underwent a living unrelated renal transplantation. The donor, a 29-year-old woman, underwent laparoscopic nephrectomy, which was followed by cold ischemia for approximately...
- 20From: Pediatric Nephrology. (Vol. 26, Issue 2) Peer-ReviewedSirs, We report remarkable symptoms of Wegener's granulomatosis (WG), which might occur more often than reported so far. Two patients (13 and 16 years old) with acute renal failure due to WG developed simultaneous...